For researchers

Is your paper here?

2,663 papers hold this site up. Every variant and condition page names the research it was written from, and this is that list, gathered from the pages themselves. Search your name, your journal, or a PMID.

12 matches for Hunt SE.

Genome-wide meta-analysis identifies 11 new loci for anthropometric traits and provides insights into genetic architecture
Nature genetics · 2013 · PMID 23563607 · open access
12 pages+
rs2207139 rs11205303 rs798554 rs6662509 rs1885486 rs2280470
Analysis of immune-related loci identifies 48 new susceptibility variants for multiple sclerosis
International Multiple Sclerosis Genetics Consortium (IMSGC) · Wellcome Trust Case Control Consortium 2 (WTCCC2) and 1 more consortia
Nature genetics · 2013 · PMID 24076602 · open access
12 pages+
rs35967351 rs2726518 rs1843938 rs7120737 rs12296430 rs4772201
Genetic risk and a primary role for cell-mediated immune mechanisms in multiple sclerosis
International Multiple Sclerosis Genetics Consortium · Wellcome Trust Case Control Consortium 2
Nature · 2011 · PMID 21833088 · open access
11 pages
rs11154801 rs12212193 rs7522462 rs9282641 rs10466829 rs2119704
Large-scale association analysis provides insights into the genetic architecture and pathophysiology of type 2 diabetes
Wellcome Trust Case Control Consortium · Meta-Analyses of Glucose and Insulin-related traits Consortium (MAGIC) Investigators and 4 more consortia
Nature genetics · 2012 · PMID 22885922 · open access
10 pages
rs7640539 rs17168486 rs13405158 rs11063069 rs11709077 rs2075423
Genome-wide association analysis identifies 13 new risk loci for schizophrenia
Multicenter Genetic Studies of Schizophrenia Consortium · Psychosis Endophenotypes International Consortium and 1 more consortia
Nature genetics · 2013 · PMID 23974872 · open access
10 pages
rs6461049 rs1006737 rs17691888 rs17504622 rs778371 rs11995572
Genome-wide trans-ancestry meta-analysis provides insight into the genetic architecture of type 2 diabetes susceptibility
DIAbetes Genetics Replication And Meta-analysis (DIAGRAM) Consortium · Asian Genetic Epidemiology Network Type 2 Diabetes (AGEN-T2D) Consortium and 3 more consortia
Nature genetics · 2014 · PMID 24509480 · open access
9 pages
rs17791513 rs6813195 rs17106184 rs849135 rs2261181 rs9936385
High-density genetic mapping identifies new susceptibility loci for rheumatoid arthritis
Biologics in Rheumatoid Arthritis Genetics and Genomics Study Syndicate · Wellcome Trust Case Control Consortium
Nature genetics · 2012 · PMID 23143596 · open access
4 pages
rs13397 rs13426947 rs59466457 rs2834512
Multiple common variants for celiac disease influencing immune gene expression
Nature genetics · 2010 · PMID 20190752 · open access
3 pages
rs2327832 rs10806425 Celiac Disease
Interaction between ERAP1 and HLA-B27 in ankylosing spondylitis implicates peptide handling in the mechanism for HLA-B27 in disease susceptibility
Spondyloarthritis Research Consortium of Canada (SPARCC) · Australo-Anglo-American Spondyloarthritis Consortium (TASC) and 1 more consortia
Nature genetics · 2011 · PMID 21743469
3 pages
rs11249215 rs11616188 Ankylosing Spondylitis
Dense genotyping of immune-related disease regions identifies 14 new susceptibility loci for juvenile idiopathic arthritis
Boston Children's JIA Registry · British Society of Paediatric and Adolescent Rheumatology (BSPAR) Study Group and 7 more consortia
Nature genetics · 2013 · PMID 23603761 · open access
2 pages
rs10174238 rs72698115
Identification of new susceptibility loci for osteoarthritis (arcOGEN): a genome-wide association study
arcOGEN Consortium · arcOGEN Collaborators
Lancet (London, England) · 2012 · PMID 22763110 · open access
1 page
rs11177
Common variants in the HLA-DRB1-HLA-DQA1 HLA class II region are associated with susceptibility to visceral leishmaniasis
LeishGEN Consortium · Wellcome Trust Case Control Consortium 2
Nature genetics · 2013 · PMID 23291585 · open access
1 page
rs9271858

Questions about this list

Why is my paper here? I never gave you permission.

Nothing of your paper is reproduced here. This page records that a MyGeneLog™ page cites your work, and links to the paper on PubMed — which is what a citation is. Your title and author list come from Europe PMC, the same public record a library catalogue uses.

Why are all the authors listed instead of "et al."?

Because a study that needed 622 people is a different kind of study from one that needed four, and "et al." erases that. Twelve names show and the rest are one click away. Everyone is searchable, including the 187th name on a consortium paper — that person exists and may be looking for it.

My name is misspelt, or the paper is wrong.

Tell us and it is fixed. Names come from the public record and initials are inconsistent across databases; where two people share a form of a name we cannot separate them, and we would rather be corrected than guess.

My paper is not here. Does that mean you rejected it?

No. This catalogue is assembled from what a small team could read, and it is thinnest outside European-ancestry cohorts. A paper is absent because nobody has got to it, not because it was assessed and turned down.

Which pages rest on a paper?

Each row lists them, up to six, and links to each one. The count beside a row is how many pages cite it in total.

How often does this change?

It is rebuilt from the pages every hour. A paper appears the moment a page starts resting on it and disappears if that page stops — there is no separate list to keep in step, because the citation lives on the page that makes the claim.