For researchers

Is your paper here?

2,646 papers hold this site up. Every variant and condition page names the research it was written from, and this is that list, gathered from the pages themselves. Search your name, your journal, or a PMID.

8 matches for Flannick J.

Discovery and systematic characterization of risk variants and genes for coronary artery disease in over a million participants
Biobank Japan · EPIC-CVD and 1 more consortia
Nature genetics · 2022 · PMID 36474045 · open access
12 pages+
rs2215614 rs72836800 rs283485 rs6088595 rs12952244 rs4790881
Genome-wide association studies in the Japanese population identify seven novel loci for type 2 diabetes
Nature communications · 2016 · PMID 26818947 · open access
6 pages
rs1116357 rs1575972 rs9309245 rs67156297 rs7107784 rs78627331
Refining the accuracy of validated target identification through coding variant fine-mapping in type 2 diabetes
ExomeBP Consortium · MAGIC Consortium and 1 more consortia
Nature genetics · 2018 · PMID 29632382 · open access
6 pages
rs16826069 rs1127787 rs7572857 rs2233580 rs56200889 rs35658696
Re-analysis of public genetic data reveals a rare X-chromosomal variant associated with type 2 diabetes
Nature communications · 2018 · PMID 29358691 · open access
4 pages
rs1708302 rs2943656 rs71320321 rs115918645
The First Genome-Wide Association Study for Type 2 Diabetes in Youth: The Progress in Diabetes Genetics in Youth (ProDiGY) Consortium
ProDiGY Consortium
Diabetes · 2021 · PMID 33479058 · open access
3 pages
rs72982988 rs10992863 rs113748381
A Loss-of-Function Splice Acceptor Variant in <i>IGF2</i> Is Protective for Type 2 Diabetes
Diabetes Prevention Program Research Group · Broad Genomics Platform and 2 more consortia
Diabetes · 2017 · PMID 28838971 · open access
1 page
rs149483638
Exome sequencing of 20,791 cases of type 2 diabetes and 24,440 controls
Broad Genomics Platform · DiscovEHR Collaboration and 8 more consortia
Nature · 2019 · PMID 31118516 · open access
1 page
rs11257600
Genome-wide discovery and integrative genomic characterization of insulin resistance loci using serum triglycerides to HDL-cholesterol ratio as a proxy
Nature communications · 2024 · PMID 39277575 · open access
1 page
rs61905108

Questions about this list

Why is my paper here? I never gave you permission.

Nothing of your paper is reproduced here. This page records that a MyGeneLog™ page cites your work, and links to the paper on PubMed — which is what a citation is. Your title and author list come from Europe PMC, the same public record a library catalogue uses.

Why are all the authors listed instead of "et al."?

Because a study that needed 622 people is a different kind of study from one that needed four, and "et al." erases that. Twelve names show and the rest are one click away. Everyone is searchable, including the 187th name on a consortium paper — that person exists and may be looking for it.

My name is misspelt, or the paper is wrong.

Tell us and it is fixed. Names come from the public record and initials are inconsistent across databases; where two people share a form of a name we cannot separate them, and we would rather be corrected than guess.

My paper is not here. Does that mean you rejected it?

No. This catalogue is assembled from what a small team could read, and it is thinnest outside European-ancestry cohorts. A paper is absent because nobody has got to it, not because it was assessed and turned down.

Which pages rest on a paper?

Each row lists them, up to six, and links to each one. The count beside a row is how many pages cite it in total.

How often does this change?

It is rebuilt from the pages every hour. A paper appears the moment a page starts resting on it and disappears if that page stops — there is no separate list to keep in step, because the citation lives on the page that makes the claim.