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Stands on its own.
Nothing else here links to this position yet — but the page above is the point: what the
research found, what each genotype means, and where it came from, in language you can
read. Links to conditions, drugs and the senses appear automatically if we write them.
What the study found
Who was studied 348,591 European ancestry individuals, 6,000 African ancestry individuals, 9,116 Asian ancestry individuals, 7,505 individuals.
The effect
Each copy of the A allele shifted the measure 0.0177 nmol/l lower (95% confidence interval 0.011-0.024); p = 3 × 10−12.
Where it sits Chromosome 16, band 16q22.1 — in an intron of SMPD3.
What each result means
A/ATwo copies of the version the research linked to Lipoprotein (a) levels. That is the strongest link of the three results. A link in a study is not a diagnosis.
A/GOne copy of the version the research linked to Lipoprotein (a) levels. That is a smaller link than two copies.
G/GNo copy of the version the research linked to Lipoprotein (a) levels. This is the starting point the study compared everyone against.
Arteriosclerosis, thrombosis, and vascular biology · 2021 · PMID 33730874
Questions about rs1110572
What is rs1110572?
rs1110572 is a single position in the genome, in or near the PRMT7 gene. Published research associates it with lipoprotein (a) levels. A single variant does not decide an outcome — it shifts a probability, and for most common variants the shift is small.
Does having rs1110572 mean I will get this?
No. Common variants like this one move a probability slightly and, on their own, rarely decide anything about one person. Nothing on this page is a diagnosis, and health decisions should be made with a clinician who can see your whole picture.
Where does the information about rs1110572 come from?
GWAS Catalog, Arteriosclerosis, thrombosis, and vascular biology 2021, PMID:33730874. Every variant on MyGeneLog comes from public primary sources and is published with its citation and the date it was checked.
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